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the spandex material around the frame to give it a bit of stretch, allowing a bit more light from the video screen to pass through it. Now, your mirror can be painted to look more like an official tool of witchcraft and then placed in front of your video screen on a table top for your Halloween visitors. The video screen is simply covered up with a bit of dark cloth and then surrounded by various objects, such as candles or other interesting objects that will keep attention focused into the center of the screen. If you like to go all-out on Halloween, then a false wall that will allow the ghost mirror to be hung over a video screen opening will make the effect look very realistic, fooling many who encounter the mirror. A simple false wall can be made from a

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of movement by virtue of its connections, via thalamocortical bers, with the corticospinal system and other descending cortical pathways 5 Several other parts of the cerebral cortex, particularly the premotor and supplementary motor cortices These structures are involved in the programming (ie, the sequencing and modulation) of voluntary movement 6 The prefrontal cortex These parts are involved in the planning and initiation of willed movement Fibers from the prefrontal cortex project to the supplementary and premotor cortex and provide the input to these more strictly motor areas Similarly, certain parietal cortical areas (superior parietal lobule) supply the somatic sensory information that activates the premotor and supplementary motor cortices and leads to directed movement In addition, other parts of the nervous system concerned with tactile, visual, and auditory sensation are connected by ber tracts with the motor cortex These association pathways provide their own sensory regulation of motor function.

The impairments of motor function that result from lesions in these various parts of the nervous system may be subdivided into (1) paralysis due to an interruption of lower motor neurons, (2) paralysis due to dysfunction of upper motor (corticospinal) neurons, (3) apraxic or nonparalytic disturbances of purposive movement due to involvement of association pathways in the cerebrum, (4) involuntary movements and abnormalities of posture due to disease of the basal ganglia, and (5) abnormalities of coordination (ataxia) due to lesions in the cerebellum The rst two types of motor disorder and the apraxic disorders of movement are discussed in Chap 3; extrapyramidal motor abnormalities are discussed in Chap 4; and disorders of coordination, in Chap 5 A miscellaneous group of movement disorders tremor, myoclonus, spasms, and tics and disorders of stance and gait are considered in Chaps 6 and 7.

Impairment or loss of motor function due to primary disease of peripheral nerves and striated muscle or to a failure of neuromuscular transmission forms the subject matter of Chaps 46 to Chap 55..

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